DNA replication stress: a source of APOBEC3B expression in breast cancer

Abstract APOBEC cytidine deaminases have been implicated as major contributors to the mutation burden in many cancers on the basis of their mutational signature. A new experimental study sheds light on the inciting factors, linking APOBEC3B expression to oncogene- and drug-induced replication stress.

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PID https://www.doi.org/10.6084/m9.figshare.c.3636500.v1
URL https://dx.doi.org/10.6084/m9.figshare.c.3636500.v1
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Author Cescon, David
Author Haibe-Kains, Benjamin
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Collected From Datacite
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Publication Date 2016-12-15
Publisher Figshare
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keyword FOS: Health sciences
keyword FOS: Biological sciences
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Source https://science-innovation-policy.openaire.eu/search/dataset?datasetId=scholix_____::c6fe1ccb0b039c77b39d88f0f7749091
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Last Updated 11 January 2021, 14:26 (CET)
Created 11 January 2021, 14:26 (CET)